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1. Li, C-Y., Yandell, D.W., and Little, J.B.(1992) Molecular Mechanisms of Spontaneous and Induced Loss of Heterozygosity in Human Cells In Vitro. Somatic Cell and Molecular Genetics, 18: 77-87
2. Li,
C-Y.,
Yandell, D.W., and Little, J.B. (1992)
Evidence for Coincident Mutations in Human Lymphoblast Clones
Selected for Functional Loss of a Thymidine Kinase Gene. Molecular
Carcinogenesis, 5:270-277
3. Spiro, I. J., Yandell,
D.W., Li, C-Y.
, Saini, S., Ferry, J., Powelson J., Katkov, W.N., and Cosimi,
A.B (1993). Lymphoma of Donor Origin
Occuring at the Porta Hepatis of Transplanted Liver. New England
Journal of Medicine, 329:27-29
4. Poremba, C., Dockhorn-Dworniczak, B., Merrit, V., Li,
C-Y. (1993) Heidle, G., Tauber,
P., Bocker, W., , and Yandell, D.W. Immature Teratomas of
Different Origin Carried by a Pregnant Mother and Her Fetus.
Diagnostic Molecular Pathology , 2:131-136
5. Li, C-Y., Yandell, D.W., and
Little, J.B. (1994) Elevated frequency
of Microsatellite Mutations in Human Lymphoblast Clones Selected
for Mutations at Thymidine Kinase locus. Molecular & Cellular
Biology , 14:4373-4379
6. Li, C-Y., Nagasawa, H., Tsang,
N., and Little, J.B. (1995) Radiation-Induced
Irreversible G0/G1 Block is Abolished in Human Diploid Fibroblasts
Transfected with the Human Papilloma Virus E6 gene: Implication
of the p53-Cip1/WAF1 pathway. International Journal of Oncology,
1 : 223-236
7. Li, C-Y., Suardet, L., and
Little, J.B. (1995) Potential Role
of WAF1/Cip1/p21 as a Mediator of TGF-b Cytoinhibitory Effect
. Journal of Biological Chemistry 270:4971-4974
8. Nagasawa, H., Li, C-Y., Maki,
C.G. , Imrich A. and Little, J.B. (1995)
Relationship between Radiation-Induced G1 Phase Arrest and
p53 Function in Human Tumor Cells Cancer Research 55:1842-1846.
9. Little, J.B. , Nagasawa, H., Keng, P., and Li,
C-Y. Absence of Radiation-induced G1-arrest in Two
Closely Related Human Lymphoblast Cell Lines That Differ in
p53 Status Journal of Biological Chemistry 270:11033-11036.
10. Tsang, N., Nagasawa, H., Li, C-Y.,
and Little, J.B. Abrogation of p53 function by Transfection
of HPV16 E6 Gene Enhances the Resistance of Human Diploid
Fibroblasts to Ionizing Radiation Oncogene 10:2403-2408.
11. Li, C-Y., Nagasawa, H., Dahlberg,
W.A., and Little, J.B. (1995) Published
Capacity for p53 in Mediating a Radiation-Induced G1 Arrest
in Established Human Tumor Cell Lines Oncogene :1885-1892.
12. Little, J.B., Li, C-Y., Nagasawa,
H., Pfenning, T., and Vetrovs, H. Genomic Instability and
Radiation Mutagenesis (1996) J. Chimie
Phys. 93:157-164
13. Li, C-Y., Nagasawa, H., Dahlberg,
W.A., and Little, J.B. (1996) The
Role of Tumor Suppressor Genes in Determining a Radation-induced
G1-arrest and Human Cell Carcinogenesis Radiation Oncology
Investigations, 3:268-271.
14. Huang, H-M., Li, C-Y, Little,
J.B. (1996) Abrogation of p53 Function
in Human Tumor Cells Does Not Alter Their Sensitivity to Ionizing
Radiation. International Journal of Radiation Biology, 70:151-160.
15. Suardet, L., Li, C-Y., and
Little J.B. (1996) Radio-induced
modulation of transforming growth factor sensitivity in a
p53 wild-type human colorectal cancer cell line . International
Journal of Cancer 68:126-31.
16. Yu, Y., Li, C-Y., Little,
J.B. (1997) Abrogation of p53 function
by HPV16 E6 gene delays apoptosis and enhances mutagenesis
but does not alter radiosensitivity in TK6 human lymphoblastoid
cells. Oncogene, 14:1661-1667.
17. Huang, Q., Shan, S, Braun, R.D., Lanzen, J., Anyrhambatla,
G., Kong, G., Borelli, M., Corry, P., Dewhirst, M.W., and
Li, C-Y. (1999)
GFP based non-invasive, in vivo monitoring of gene expression.
Nature Biotechnology, 17: 1033-1035.
18. Li, C-Y., Shan, SQ., Huang,
Q., Braun, R.D., Lanzen, J., Turnage, J., Dewhirst, M.W. (2000)
Initial stages of tumor cells-induced angiogenesis:evaluation
via skin window chambers in rodent models Journal of the National
Cancer Institute, 92:143-127
19. Huang,Q., Hu, K., Lohr, F., Zhang,L., Braun, R., Lanzen,
J., Little,JB., Dewhirst, MW., and Li,
C-Y. (2000) Heat-induced Gene
Expression as a Novel Targeted Cancer Gene Therapy Strategy
Cancer Research, In Press.
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